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高盐饮食在小鼠诱发类抑郁症状
#医学

南京医科大学的研究人员在《The Journal of Immunology》期刊上报告,高盐饮食(HSD)通过诱导名为 IL-17A 的蛋白质的产生,在小鼠身上诱发了类似抑郁的症状。在人类临床研究中,这种蛋白质已经被确定为抑郁症的诱因。研究人员还发现一种称为γδT细胞(γδT细胞)的免疫细胞是 HSD 喂养小鼠中IL-17A的重要来源,占IL-17A产生细胞的40%左右。消耗γδT细胞显著缓解 HSD 诱导的抑郁症状,确定了另一种可能的治疗方法。HSD 已经是一个重要的公共卫生问题,因为它与心血管、自身免疫和神经发散性疾病有关。此外重度抑郁症也是一个重大的公共卫生问题,其终生患病率为 15-18%。长期以来,HSD 一直与抑郁症的发生和发展有关,但其在抑郁症病因中的作用尚不清楚。在这项研究中,小鼠被喂食正常饮食或 HSD 5周,这是研究过量饮食盐摄入的常用时间框架。五周后,与正常饮食的小鼠相比,喂食 HSD 的小鼠在各种情况下对探索的兴趣减少,更不活跃,这表明小鼠出现了类似抑郁的症状。
doi: 10.1093/jimmun/vkaf019 High-salt diet drives depression-like behavior in mice by inducing IL-17A production
Ebiotrade 高盐饮食诱导小鼠出现抑郁样症状



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高盐饮食在小鼠诱发类抑郁症状
#医学

南京医科大学的研究人员在《The Journal of Immunology》期刊上报告,高盐饮食(HSD)通过诱导名为 IL-17A 的蛋白质的产生,在小鼠身上诱发了类似抑郁的症状。在人类临床研究中,这种蛋白质已经被确定为抑郁症的诱因。研究人员还发现一种称为γδT细胞(γδT细胞)的免疫细胞是 HSD 喂养小鼠中IL-17A的重要来源,占IL-17A产生细胞的40%左右。消耗γδT细胞显著缓解 HSD 诱导的抑郁症状,确定了另一种可能的治疗方法。HSD 已经是一个重要的公共卫生问题,因为它与心血管、自身免疫和神经发散性疾病有关。此外重度抑郁症也是一个重大的公共卫生问题,其终生患病率为 15-18%。长期以来,HSD 一直与抑郁症的发生和发展有关,但其在抑郁症病因中的作用尚不清楚。在这项研究中,小鼠被喂食正常饮食或 HSD 5周,这是研究过量饮食盐摄入的常用时间框架。五周后,与正常饮食的小鼠相比,喂食 HSD 的小鼠在各种情况下对探索的兴趣减少,更不活跃,这表明小鼠出现了类似抑郁的症状。
doi: 10.1093/jimmun/vkaf019 High-salt diet drives depression-like behavior in mice by inducing IL-17A production
Ebiotrade 高盐饮食诱导小鼠出现抑郁样症状

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